8-Bromo-cGMP sodium (1 µM-0.1 mM) can inhibit acetylcholine-induced increases in intracellular calcium concentrations.[1]
8-Bromo-cGMP sodium (1-100 μM; 16 h) induces the synthesis of HO-1 protein in a concentration-dependent fashion.[2]
8-Bromo-cGMP sodium (1-100 μM; 8 h) increases the resistance of LLC-PK1 cells to CsA toxicity in concentration-dependently.[2]

8-Bromo-cGMP sodium (0.3, 1, 3 nM; intrathecal administration; 10 min before the test; male ICR mice) significantly increases the tail-flick latency in Vincristine-treated mice to the level observed in vehicle-treated naive mice in dose-dependently.[3]
8-Bromo-cGMP sodium (10 mg/kg; iv; single dose) results in vasodilator responses in WT littermates and eNOS-Tg mice in C57BL/6 background (19-35 g).[4]