Osajin 对六种人类癌细胞系(包括肾癌、肺癌、前列腺癌、乳腺癌、黑色素瘤和结肠癌细胞)均表现出生长抑制活性。
Osajin 以剂量依赖性方式显著降低人类鼻咽癌细胞的存活率。
Osajin 通过多种凋亡通路诱导人类鼻咽癌细胞凋亡，包括外源性死亡受体通路，以及依赖线粒体和内质网应激的内源性通路[1][2]。

The cardioprotection provided by osajin and pomiferin treatment results from the suppression of oxidative stress and this correlates with improved ventricular function. Osajin and pomiferin attenuate the myocardial dysfunction provoked by ischemia-reperfusion. This is confirmed by an increase in both antioxidant enzyme values and total antioxidant activity[3].