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Anti-EphA1 Antibody (3F290) 是一种 Rabbit 抗体,靶向 EphA1。Anti-EphA1 Antibody (3F290) 可用于 ELISA。
Anti-EphA1 Antibody (3F290) 是一种 Rabbit 抗体,靶向 EphA1。Anti-EphA1 Antibody (3F290) 可用于 ELISA。
产品描述 | Anti-EphA1 Antibody (3F290) is a Rabbit antibody targeting EphA1. Anti-EphA1 Antibody (3F290) can be used in ELISA. |
Ig Type | Monoclonal Rabbit IgG |
克隆号 | 3F290 |
交叉反应 | Mouse |
应用 | ELISA |
推荐剂量 | ELISA: 1:5000-1:10000 |
抗体种类 | Monoclonal |
宿主来源 | Rabbit |
构建方式 | This antibody was obtained from a rabbit immunized with purified, recombinant Mouse EPHA1 / EPH receptor A1 (rM EPHA1 / EPH receptor A1; TMPY-02520; Q60750; Met1-Glu548). |
纯化方式 | Protein A |
性状 | Liquid |
缓冲液 | 0.2 μm filtered solution in PBS |
研究背景 | EPHA1 or EPH receptor A1 belongs to the ephrin receptor subfamily of the protein-tyrosine kinase family. Receptors in the EPH subfamily typically have a single kinase domain and an extracellular region containing a Cys-rich domain and 2 fibronectin type III repeats. An important role of Eph receptors and their ligands ephrins is to mediate cell-contact-dependent repulsion. Eph receptors and ephrins also act at boundaries to channel neuronal growth cones along specific pathways, restrict the migration of neural crest cells, and via bidirectional signaling prevent intermingling between hindbrain segments. Eph receptors and ephrins can also trigger an adhesive response of endothelial cells and are required for the remodeling of blood vessels. Eph receptors and ephrins have emerged as key regulators of the repulsion and adhesion of cells that underlie the establishment, maintainence, and remodeling of patterns of cellular organization. The ephrins and Eph receptors are implicated as positional labels that may guide the development of neural topographic maps. |
偶联 | Unconjugated |
免疫原 | Recombinant Mouse EPHA1 / EPH receptor A1 protein (TMPY-02520) |
抗原种属 | Mouse |
储存方式 | Store at 2°C-8°C for 1 month. Store at -20°C or -80°C for 12 months. Avoid repeated freeze-thaw cycles. Preservative-Free. |
运输方式 | Shipping with blue ice. |
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