Cat. No. | Product Name | Target | Signaling Pathways |
---|---|---|---|
T23861 |
CAY10526
CAY-10526,BTH,CAY 10526 |
Prostaglandin Receptor | GPCR/G Protein; Immunology/Inflammation |
CAY10526 (BTH) 是 mPGES-1 的选择性抑制剂,可抑制 NF-κB 信号通路。 | |||
T16856 |
SB228357
|
5-HT Receptor | GPCR/G Protein; Neuroscience |
SB228357 是一种有效且的选择性 5-HT 受体拮抗剂,对 5-HT2A、5-HT2B 和 5-HT2C 的 pKis 分别为 6.9、8.0 和 9.0。 | |||
T1955 |
SB225002
|
CXCR | Autophagy; GPCR/G Protein; Immunology/Inflammation |
SB225002 是一种有效的选择性 CXCR2 拮抗剂,抑制白介素 IL-8 与 CXCR2 的结合,IC50为 22 nM。 | |||
T12851 |
SB290157 trifluoroacetate
|
Complement System | Immunology/Inflammation |
SB290157 trifluoroacetate 是一种选择性 C3a 受体拮抗剂,IC50值为200 nM。 | |||
T6657 |
SB273005
|
Integrin | Cytoskeletal Signaling |
SB273005 是一种有效的整合素抑制剂,对 αvβ3 受体和 αvβ5 受体的 Ki 分别为 1.2 nM 和 0.3 nM。 | |||
T4674 |
SB297006
SB 297006 |
CCR | Immunology/Inflammation; Microbiology/Virology |
SB297006 是一种CCR3拮抗剂,IC50为39 nM。它能够显著抑制 CCL11 处理的神经祖细胞的增殖和神经球形成。 | |||
T75013 | SB26019 | ||
SB26019 是一种有效的抗神经炎症剂。SB26019 通过诱导单体 α-微管蛋白 (α-tubulin) 形成来调节 NF-κB 激活。SB26019 诱导的 α-微管蛋白单体抑制 p65 易位。 | |||
T28683 |
SB234551
SB 234551,SB-234551 |
||
SB234551 is a selective antagonist of endothelin ETA receptor. | |||
T28688 |
SB2602
SB-2602,SB 2602 |
||
SB2602 selectively inhibits mTOR. | |||
T16857 |
SB269652
|
Others | Others |
SB269652 is the first drug-like allosteric modulator of the dopamine D2 receptor. | |||
T28685 |
SB236057
SB-236057,SB 236057 |
||
SB236057 is a potent and selective inverse agonist for the 5-HT receptor, acting especially at 5-HT autoreceptors on nerve terminals. It produces a rapid increase in 5-HT levels in the brain. SB236057 also acts as a potent teratogen, producing severe musc | |||
T28687 |
SB251023
SB-251023,SB 251023 |
||
SB251023 is an agonist of β3-adrenoceptor. | |||
T82961 |
ASB20123
|
||
ASB20123 是一款融合了CNP/ghrelin序列的嵌合肽,具有促进骨骼生长的效能,适用于研究生长障碍和侏儒症。 | |||
T28681 |
SB220025
SB-220025,SB 220025 |
||
SB220025, a specific and potent inhibitor of human p38 mitogen-activated protein kinase, reduces the production of tumor necrosis factor induced by lipopolysaccharide. |