Cat. No. | Product Name | Target | Signaling Pathways |
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TP1756 |
Brain Natriuretic Peptide (1-32), rat
Brain Natriuretic Peptide (BNP) (1-32), rat |
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Brain Natriuretic Peptide (BNP) (1-32), rat is a 32 amino acid polypeptide secreted by the ventricles of the heart in response to excessive stretching of heart muscle cells (cardiomyocytes).Brain natriuretic peptide (type B natriuretic peptide) was origin | |||
TP1207 |
Brain Natriuretic Peptide-45, rat
BNP-45 (rat),Brain natriuretic peptide-45 rat |
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BNP-45 (rat) is a circulating form of rat brain natriuretic peptide isolated from rat heart with potent hypotensive and natriuretic potency. | |||
TP1209 |
BNP (1-32), rat TFA (133448-20-1 free base)
Brain Natriuretic Peptide (BNP) (1-32), rat TFA,BNP (1-32), rat TFA |
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Acts as a cardiac hormone with a variety of biological actions including natriuresis, diuresis, vasorelaxation, and inhibition of renin and aldosterone secretion. It is thought to play a key role in cardiovascular homeostasis. Helps restore the body\'s sa | |||
TP1206 |
BNP-45 (rat) (TFA) (123337-89-3 free base)
BNP-45 (rat) (TFA),Brain natriuretic peptide-45 rat TFA |
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BNP-45 (rat) TFA is a circulating form of rat brain natriuretic peptide isolated from rat heart with potent hypotensive and natriuretic potency. | |||
T82823 |
Brain Natriuretic Peptide-45, rat TFA
BNP-45, rat TFA |
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Brain Natriuretic Peptide-45, rat TFA (BNP-45, rat TFA) 为来源于大鼠心脏的脑利钠肽循环同源物,具有显著的降压和利尿效果。 | |||
T35832 |
Brain Natriuretic Peptide (1-32), rat acetate
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Brain Natriuretic Peptide (1-32), rat acetate (BNP (1-32), rat acetate) is a 32 amino acid polypeptide hormone synthesized by ventricular cardiomyocytes as a result of myocardial cell stretching (cardiomyocyte distension)[1]. | |||
T83905 |
Brain Natriuretic Peptide-32 (rat) acetate
BNP (14-45),B-type Natriuretic Peptide-32,Brain Natriuretic Polypeptide-32,BNP-32 |
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BNP-32(Brain natriuretic peptide-32)是心血管激素前体proBNP的肽段,能够结合大鼠血管平滑肌上的利钠肽受体(IC50 = 7.3 nM),并在同样的细胞中促进cGMP积累(EC50 = 170 nM)。BNP-32还能使经去甲肾上腺素预收缩的孤立大鼠主动脉条带松弛(EC50 = 6.7 nM),当以3 µg/kg的剂量给药时,能增加大鼠的尿量和尿液电解质排泄,并诱导低血压。 |