Cat. No. | Product Name | Target | Signaling Pathways |
---|---|---|---|
T28901 |
T-2007
5,5-Diphenylbarbituric acid,DPB |
||
T-2007 is an α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptor antagonist and gamma-aminobutyric acid (GABA) receptor agonist. T2007 and its analogs upregulates CYP3A4 and MDR1. T-2007 depressed both the degree and the duration of postt | |||
T31569 |
DP-b99
DP-b-99,DP-BAPTA-99,DPb-99,DPBAPTA-99,DP-b 99 |
||
DP-B 99 (DP-B 99, DP-BAPTA-99, DPB-99, DP-BAPTA-99) is a potential chelating agent for the treatment of acute pancreatitis. | |||
T11092 |
DPBQ
ZINC1620467,2,3-Diphenylbenzo[g]quinoxaline-5,10-dione |
p53 | Apoptosis |
DPBQ (ZINC1620467) 是一种 p53 的激活剂。DPBQ 以多倍体特异性方式激活并诱导凋亡 (apoptosis),但不会抑制拓扑异构酶或结合 DNA。 | |||
T31570 |
DPBX-L-Dopa
DPBX L-Dopa |
||
DPBX-L-Dopa is a boron-containing dopa-derivative, acting as a bladder relaxant through non-catecholamine receptors. | |||
T74438 |
β-Glucuronide-dPBD-PEG5-NH2 TFA
|
||
β-Glucuronide-dPBD-PEG5-NH2 TFA 是一种通过β-葡糖苷酸与pyrrolobenzodiazepine形成的二聚体,能够与异戊二烯化的抗体结合,生成抗体-活性分子偶联物(ADC) cIRCR201-dPBD。该化合物中的β-葡糖苷酸键作为可降解的ADC Linker,有助于减少副作用。β-Glucuronide-dPBD-PEG5-NH2 TFA 作为cIRCR201-dPBD的前体,显示出能诱导细胞凋亡(apoptosis)和阻滞细胞周期,具备抗肿瘤活性。 | |||
T74437 | β-Glucuronide-dPBD-PEG5-NH2 | ||
β-Glucuronide-dPBD-PEG5-NH2 是由 β-葡糖苷酸与 pyrrolobenzodiazepine 通过二聚体形成方式连接,并能与异戊二烯化抗体结合形成抗体-活性分子偶联物 (ADC) cIRCR201-dPBD 的化合物。其所含β-葡糖苷酸键为一种可降解 (cleavable) 的 ADC Linker,作为 cIRCR201-dPBD 的前体能有效减少副作用。此外,β-Glucuronide-dPBD-PEG5-NH2 还具备诱导细胞凋亡 (apoptosis) 和阻滞细胞周期的能力,显示出抗肿瘤活性。 |